Vaccine makers ditch control groups to hide growing catalogue of death and injury.
Tap News / Tapestry
- Tim Zook’s last post on Facebook brimmed with optimism. “Never been so excited to get a shot before,” he wrote on Jan. 5, above a photo of the Band-Aid on his arm and his COVID-19 vaccination card. “I am now fully vaccinated after receiving my 2nd Pfizer dose.”Zook, 60, was an X-ray technologist at South Coast Global Medical Center in Santa Ana. A couple of hours later, he had an upset stomach and trouble breathing. By 3:30 p.m. it was so bad his colleagues at work walked him to the emergency room. “Should I be worried?” his wife, Rochelle, texted when she got the news. “No, absolutely not,” he texted back. “Do you think this is a direct result of the vaccine?” she typed. “No, no,” he said. “I’m not sure what. But…
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As reported in the barrister’s blog:
On 10 October, the court finally discharged the guardian from his duties in relation to Karina.
Karina has now finally regained control of her own health and financial affairs.
This sounds like me: “I explain that for me walking to the bathroom raises my heart rate and is exercise for me. I also explain that as I rest sufficiently, my capacity to do more activity and exercise increases.”
Comments welcome.
via How Did You Get Others to Understand That Activity Makes You Worse?
via THIS is Lyme
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Spirochetes disseminate to the lymph nodes, bone marrow, spleen and brain within a week of infection (1).
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Lymph node germinal centers, where B cells are supposed to mature and be assigned an immune system function, are rendered incompetent (2).
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Meanwhile, the toxic triacyl lipoproteins that are shed by spirochetes on blebs of their outer surface get to work causing tolerance and cross tolerance (2,3,4), AKA shutting down the immune system (5,6).
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There is generalized immune suppression at the same time there are brain inflammation and neurologic complications (7,8,9,10).
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Opportunistic infections take hold and herpes viruses reactivate (11,12).
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Half the cases don’t recover fully, regardless of treatment (13,14,15).
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The outcome is cancer-like (16,17).
please visit the original post to double-check that the references are accurate and from professional sources
I have been living under the illusion that our Government’s would protect us, the people. Now there is proof that Lyme Disease patients have been lied to by their supposed allies. The ILADS antibiotic protocol that I was so desperate to apply is not even capable of curing Lyme Disease or any other spirochaetal disease so I am not happy having put myself through 12 months of horrific herxheimer reactions, side effects and further weakening of my immune system. That was back in 2012- 2013. I have more symptoms now than I did before treatment which has got to indicate failure of the antibiotic protocol. The latest thing to add to my complaints is delayed gastric emptying. It’s cruel.
Follow the link at the top of the page to find out about a vaccine that was supposed to make some people very rich. But the story of Lyme does not even begin there. There is so much for you to learn in order to find out the truth and that implies time and effort on your part. You may like to start at Truthcures.org
I’ve got LymeAIDS, another B-cell disease, just like my Dad had B-cell Lymphoma. At the moment, the only thing that is stopping me from loosing my ability to walk and think is 5mg of prednisolone a day which is NOT a cure. Antibiotics would keep me going by reducing the bug load which is also not a cure, but I am not allowed to have them anyway except for one week for a hoped-for lung-infection. I say hoped-for because the changes in my lung-nodules could be something far worse (UPDATE: They took two years to resolve all by themselves). I know the antibiotic protocol works because it gave me back another couple of years of being able to act normally and participate in life. I do not know what the antibiotics worked on. I’ve had other potentially chronic infections in the past like Mycoplasma Pn. I had 12 months worth of a few mixed and rotated antibiotics and the bicillin injections were the mainstay of my treatment but I suffered so much with rebound joint pain after each injection for many days, every fortnight, during that 12 months that I have not been in a hurry to line up for more torture. Money has a lot to do with it too and a nagging doubt that the treatment that forced me to bed for a year, and which I was prepared to endure as a good sign (Herxheimer healing crisis) was actually just prolonged enforced bed-rest and that the proneness of it all did more to help my body than the antibiotic protocol designed by a local LLMD. Anyway, six years later and I feel like I am dying so I am happy to take low-dose steroids if they bring quality to my life. They have made me feel so much better in the past. (UPDATE: they worked so that I could walk properly again, but only after my home had already been redundantly fitted with half-steps and rails. It all felt miraculous but it was probably just adrenal fatigue being remedied).
No-one really knows how ill I am because I look pretty-well presented. Whether I am able to care for myself or not, the outward appearance is OK except for dark circles under my eyes which are relatively new on the scene.. My doctor thinks that a sleep study is going to reveal all the answers. Now who is the naive one? (UPDATE: This second sleep study was also “normal” as far as sleep apnea goes)
All this ME/CFS fibromyalgia Lyme stuff is all tied together with the glandular fever I got so young but there was something else that made me delirious enough to write about my hallucinations for a grade 7 Creative Writing assignment before the glandular fever and a reactivation of chicken pox after. My glands have been going up and down ever since but the doctors won’t let me be diagnosed with chronic EBV because it doesn’t exist. Lyme disease does not exist in Australia either they say. The family tick encounter was even before all that and all it seemed to have left me with were leg pains that I was told were growing pains. I don’t know any of this is for sure because it wasn’t me who had a tick bite (that I was aware of) but finally after all these years I’ve decided what matters is to stop being so naive and accept the fact that money, profits, patents and threats have more influence over people than the truth. And all that, filters down to your friendly General Practitioner (Jack-of-all trades doctor) who will not read the research even when it is handed to her.
It’s not conspiracy theory any more. It is now all documented in the charge sheets found on truthcures.org so don’t be naive and learn another possible truth before you die. The role of fungal proteins acting as antigens. This research effectively changes the focus from killing spirochaetes to dealing with consequent immune deficiency.
Please click on the photo above to also find out about so-called Lyme vaccines and deceit.
Some idiot this morning had the gall to tell me in a “private” message that the science saying that “Lyme” causes an AIDS-like disease in the majority of victims is, in his important opinion, “dead wrong.”
I am not here to argue over treatments or whether the well documented model of Borrelia-post-sepsis disease is correct. If you are a Lyme victim, and you claim that this model can’t possibly be correct, because *you* got better with antibiotics, or pot, or whatever…just leave. If you can’t have the human decency or capacity for critical thought to realize that your personal case is not equal to the disease model (i.e. the center of the universe), then have a lovely day and be well.
Just don’t run around telling people that because *you* are better (temporarily or otherwise) Lyme is not so serious as to kill people, disable children, and cause permanent immune…
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Here’s two of Thomas Grier’s posts from his Facebook page:
This photo (below) of Dr. Willy Burgdorfer and myself is from 1992 or possibly 1993. This was one of the first International Lyme Disease conferences I attended.
It might not show, but during this conference I could barely stand upright, and all the noise and activity was like a swarm of bees and geese making my brain swirl with anxiety and fear of collapsing.
In 1992 I didn’t know who the good guys or bad guys of Lyme disease research were; I didn’t even think in those terms. I tried to listen to every lecture with an open mind and acceptance because I was still naïve enough to believe that science and medicine was a pure and untainted by politics or special interests.
Barb Jones RN my best friend and Lyme patient advocate introduced me to her friend Willy Burgdorfer, and we had lunch together.
In subsequent conferences I would meet and speak with Willy over the next 20 years perhaps another dozen times; each time being special and memorable. I knew from the beginning that Willy Burgdorfer was a good guy.
The first question I asked Willy was about the species variation of Borrelia. I mentioned Relapsing Fevers and wondered if we would see the same kind of diversity of Borrelia species in America. He told me that almost every isolate in the Rocky Mountain Lab (NIH) collected was its own isolate.
“Yes. We will see diversification! Almost every tick species has its own specific Borrelia organism, this is seen even in America. We are just lucky that we do not have the African tick species like the Moubata ticks living in our woods.” He said in his thick Swiss accent.
He then went on to tell me about the great pioneers of Borrelia research and facts about Old World Relapsing Fevers and how they differed from Willy’s first isolate of Lyme disease from Shelter Island.
I mentioned that the great Nobel Prize laureate James Watson had been instrumental in showing that bacteria can exchange genetic information and plasmids and asked if Borrelia either exchanged DNA inside the ticks or the animal hosts.
“We do not know this but it is likely and needs to be addressed.” He said in a way that tried to end this conversation.
Pushing him I asked if European hard shelled ticks had ever been able to maintain a species like Borrelia duttoni when feeding on infected host animals. I asked because Dr. Dutton from Britain might have done such an experiment with his native tick?
Willy just smiled and said:
“No evidence of this has ever been published, and it is unlikely that this would be a focus of any research.”
Dr. David Dorward then joined us and he and Willy walked away. This was my first conversation with Willy, but over the years we would talk on the phone and I gained a true appreciation of how global his pursuit of microbiology was.
Willy did not think in just terms of American disease, but of global concerns.
It was clear from early on that Willy was not a supporter of the rigid thinking and black and white statements from YALE and the CDC telling us so early on that Lyme disease was contained to the NE USA. Or dogmatic maxims that Borrelia burgdorferi was only found in the Ixodes damminii ticks, or that there was no relation to any Relapsing Fever Borrelia species. How, in just a few months after isolating Borrelia burgdorferi, could any credible scientist make such fixed determinations?
Of course Willy was right. Lyme disease could not be contained or be explained in rigid unyielding terms. Lyme disease was ever changing ever moving and ever vexing.
Later I will post about a few conversations I had with other Lyme disease researchers over the past 25 years.
END
Thomas Grier (left) and Willy Burgdorfer
Thomas Grier Executive Director
Dr. Paul H Duray Pathology Research
902 Grand View Ave
Duluth MN 55812
(218)728-3914
TO: Editors of Arts and Patient magazineDear Editors: I am writing to express a concern about the article you recently published, written by Professor Jan Keppel Hesselink. The article was a position paper about how patients and the lay-public have exaggerated concerns or misrepresented the seriousness of Lyme disease and modern vaccines.
As someone who has been involved with Lyme disease and Lyme disease research since 1990, I have concerns about academics who express strong opinions on this subject, but offer little or no scientific support or research of their own to support that patients are trying to influence medicine in a negative direction.
Since 1991 I have fought for pathology based Lyme disease research to put certain myths and misinformation about Lyme-Borreliosis to rest. For over 25 years the lay-public has been told facts about Lyme disease that quite frankly are not true and were never true. Many of these untrue facts are still being propagated by major physician groups and medical institutions; but lets look at the facts from a position of human pathology. Then decide who is misleading whom?
We were told that only the Ixodes dammini tick could carry Lyme disease and that it was only found in the NE USA therefore Lyme disease was only in the NE USA. The truth is there never was an Ixodes dammini tick, Dr. Andrew Spielman of Harvard misidentified the Black Legged tick which is found throughout North America and several close cousins of this tick all carry the Lyme spirochete and B. burgdorferi is found all through the Northern Hemisphere.
We were told that Borrelia burgdorferi was never intracellular. In truth Lyme disease is very much an intracellular disease and I have attached our own research photos to support that Lyme disease enters the brain and then enter both glial cells and human neurons. We urge every country to do human-brain autopsies as we have done, to see the whole truth of this disease.
We were told for over two decades that Lyme disease is not transferred from mother to fetus. (2006 Dr. Gary Wormser Yale in Under Our Skin documentary.) Once again we have the pathology evidence from 1989 of eight fetal autopsies showing Borrelia in the umbilical fluid, in the placenta, and in almost every fetal organ at necrotopsy. (See attach images)
What else did they get wrong? (When I say they I generally mean in my opinion that the misinformation about Lyme was most damaging from those that had the most influence on the Medical Community which includes the CDC, Yale, SUNY, Harvard and Mayo Clinic, the ACP and the IDSA)
Another untrue truth we were told was that only one species of Borrelia causes Lyme disease. Did they learn nothing from Relapsing Fevers? Borrelia changes and evolves like no other bacteria. It is constantly changing and evolving. We now have 12 species that cause Lyme disease and several species that we call Lyme-Like.
Another non-fact is that we were told that there was only one reservoir host – the white footed mouse. Of course now we know many rodents and birds are carriers of the disease and transmit it to feeding ticks. The range of these animals is far beyond the NE USA where we were told was the only place Lyme disease could exist.
In the Yale medical Report by Marc Voortman it says that patients with a positive ELISA test but a negative western blot can ignore symptoms like a bull’s-eye rash, Bell’s Palsy, and swollen joints and no treatment is necessary, but if the Western Blot test is positive you can treat up-to 2 weeks with doxycycline. I have attached pathological proof that two weeks is not sufficient for late Lyme symptoms.
The list of misinformation shows that the medical experts from the very discovery of this disease got over a dozen major-critically important facts about Lyme Borreliosis completely wrong, and perpetrated misinformation long after pathology had proved them wrong.
The most damaging untruth about Lyme disease was the insistence that Lyme disease does not persist in the human body after antibiotic treatment.
Attached are photos from a patient that lives just a few miles from me. He was a logger who was sick for 30+ years. He was diagnosed with Lyme disease and treated with aggressive antibiotic combinations for over seven years. When he died we found both Borrelia burgdorferi and Borrelia miyamotoi in his brain, and we found live B.burgdorferi and B. mayoni in his testicle.
Lyme disease does persist. It gets into the brain and inside brain cells. (Attached pathology photos from brain autopsies) In addition we have cultured B. burgdorferi out of Alzheimer’s brains.
It is unfortunate to see a position paper like this in 2016 considering that this is the year that many major Universities are clamoring to be the first to publish that they have found an infectious etiology to Alzheimer’s disease, and Borreliosis is the front running contender as a brain pathogen. While we have been using pathology for over twenty-years to prove Borrelia and Alzheimer’s are related, others are just now waking up to the fact that having both a cause and a potential treatment for Alzheimers, is worthy of major recognition.
2016 is the year that the race for the Nobel Prize has begun in earnest, and the group that gets accepted recognition of Borrelia in human brain causes Alzheimer’s pathology will most likely win the prize. You will see papers by Harvard and other major Universities vying for the pole position to prove Alzheimer’s has an infectious etiology.
While others live in the past and criticize patients for their efforts to use pathology to prove their illness has been mismanaged, the medical community has moved on and accepted that the facts they once thought were true are now part of history, and that the Lyme patients were in fact more right than the experts.
Lyme disease persists in the brain, the serology tests are missing brain infections, pregnant mothers can transmit the disease to their unborn child, sexual transmission is likely now that we documented Borrelia in human testicles even after antibiotics, and Lyme disease is found through-out the Northern hemisphere caused by more than a dozen species of Borrelia and carried by many ticks, and Lyme disease can kill as it did in the New York man who had Borrelia in his heart.
So when a patient disagrees with their doctor and explains that they are still sick after treatment. Who is right? The doctor citing 30 year old dogma, or the patient that died and had a brain-autopsy done that revealed a persistent brain infection despite years of antibiotics?
Thomas M. Grier (Exec Director)
Dr. Paul H. Duray Pathology Research Foundation
